您目前的位置:院首页>首页 > Lecturer

Bian Xiling

Bian Xiling

 

Dr. Xiling Bian is currently a Lecturer in the Department of Molecular and Cellular Phaymacology at Peking University School of Pharmaceutical Science, where she is trying to identify the ion channel-targets of natural medicine and explore neurological functions of ion channels. Dr. Bian received her Ph.D. in neuroscience from Chinese Academy of Sciences in 2008 with her research topic ‘Pheromone-Processing Circuits in the Medial Amygdala’. After that, Dr. Bian entered Peking University Health Science Center. She has published four first (co-first) authored research articles in scientific journals such as Nature Communications. She received three grants, including one grant from National Natural Science Foundation of China and another one form Beijing City Youth Talent Plan. She was honored as Model Teacher of Peking University Health Science Center in 2011.

1. Xiling Bian*, Physiological and morphological characterization of GABAergic neurons in the medial amygdala, Brain Reserch. (2013); 1509:8-19

2. Limin Shi1, Xiling Bian1, Zhiqiang Qu, Zegang Ma, Yu Zhou, KeWei Wang*, Hong Jiang* & Junxia Xie*, Peptide hormone ghrelin enhances neuronal excitability by inhibition of Kv7/KCNQ channels ,NATURE COMMUNICATIONS, (2013), 4:1435 (共同第一作者)

3. Zhenyu Hu1, Xiling Bian1, Xiaoyan Liu, Yuanjun Zhua, Xiaoyi Zhang, Shizhong Chen, Kewei Wang, YinyeWanga*, Honokiol protects brain against ischemia–reperfusion injury in rats through disrupting PSD95–nNOS interaction, Brain Reserch,1491,(2013) 204-212(共同第一作者)

4. Xiling Bian, Yuchio Yanagawa, Wei R. Chen, Minmin Luo, Cortical-like functional organization of the pheromone-processing circuits in the medial amygdala, J Neurophysiol, (2008),99(1),77-86

5. Xiling Bian, Targeting voltage-gated Kv7/KCNQ/M-channel for therapeutic potential of neuropsychiatric disorders, Journal of Chinese Pharmaceutical Sciences, (2014),vol 23, pp 4-15.

6. Yeping Bi, Hui Chen, Jun Su, Xu Cao, Xiling Bian and KeWei Wang*, Visceral hyperalgesia induced by forebrain-specific suppression of native Kv7/KCNQ/M-current in mice, Molecular Pain ,2011, 7:84

7. Yi-Quan Tang, Ping Liang, Jingheng Zhou , Yanxin Lu,, Lei Lei, Xiling Bian, KeWei Wang *, Auxiliary KChIP4a Suppresses A-type K+ Current through Endoplasmic Reticulum (ER) Retention and Promoting Closed-state Inactivation of Kv4 Channels ,JOURNAL OF BIOLOGICAL CHEMISTRY, (2013),288(21), pp 14727-14741,2013/5/24